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This occurs when the sound transmission
through the ear canal (outer ear) and middle ear is blocked. Causes
of conductive hearing loss within the outer ear canal include impacted
cerumen (ear wax), drainage, foreign object (bead, q-tip), or perforated
tympanic membrane (hole in the ear drum). In most cases of middle
ear-caused conductive loss, fluid develops
within the middle ear space when the eustacian tube can not
properly ventilate the middle ear space. This is known as middle
ear infection. Other middle ear causes of hearing loss include
tissue growth or benign tumor (cholesteatoma) filling the middle
ear space, damage to the ossicular chain, and in some cases a bony
growth within the footplate of the stapes where it attaches to
the oval window of the cochlea (otosclerosis).
Most conductive hearing loss can be treated
medically or surgically. Obviously, when the blockage of the ear
canal or middle ear is removed, or the damaged portion of the middle
ear is repaired, most, if not all of the hearing is restored. In
cases of middle ear infections, pressure equalizing tubes
are inserted to allow for proper middle ear ventilation, preventing
middle ear fluid from developing again. In cases of damage or disruption
of the ossicular chain, surgery can be done, using a prosthetic device
to take the place of the ossicular chain to conduct sound properly
to the inner ear.
With
sensorineural hearing loss, the obstruction of sound transmission
occurs within the cochlea and/or auditory nerve. Sensori, or cochlear
type of hearing loss occurs when there is damage to hair cells and/or
basilar membrane of the cochlea, disrupting the chemical and electrical
transmission of sound. This damage is often due to exposure to loud
noises, or acoustic trauma. In most cases, the hair cells tuned for
high frequencies are damaged first, as they are more vulnerable to
violent sound waves due to their location on the membranes of the
cochlea. Such noise damage can also result in tinnitus (see tinnitus
section), which may accompany the hearing loss.
Cochlear hearing loss can also be caused
by inflammation of the cochlea, usually from fungal, bacterial
or viral infections. Bacterial, fungal and viral infections may
reach the cochlea through the cochlea aqueduct, or via blood supply
to the inner ear. Inflammation of the
inner ear can also cause tinnitus and dizziness along with hearing
loss, as observed in cases of Meniere’s disease (see dizziness).
Exposure to toxic chemicals, which enter the inner ear via blood
supply, can also cause hearing loss within the cochlea. Circulatory
deficiencies, such as high blood pressure, or lack of blood supply
to the inner ear, also can cause hearing loss. With lack of blood
supply comes lack of adequate oxygen and nutrients to the inner ear
to maintain its normal function. Like viral infections, circulatory
hearing loss many times is accompanied with dizziness and tinnitus.
(beyond the cochlea)
causes of hearing loss
involve the auditory nerve and/or the neurological stations and auditory
cortex of the brain. Structural changes in the retrocochlear portion
of the hearing system usually includes demyelination, or the nerve’s
loss of ability to transmit neurological impulses to the cortical
areas of the brain. However, interruption of blood supply and toxicity
can also cause neurological disturbances in hearing. In some cases,
a benign tumor known as an acoustic neuroma, can interfere with the
neural transmission of sound within the auditory nerve. This tumor
usually can be found within the internal auditory canal, where the
auditory nerve travels from the cochlea to nerve stations and cortical
areas. The tumor will press against the auditory nerve, causing hearing
loss to that affective ear. In most cases of acoustic neuroma, unilateral
hearing loss is detected (or hearing loss in one ear but not the
other).
It must be noted that the most common cause of sensorineural
hearing loss (and one that can not be avoided) is Presbycusis, or
aging. In most cases of Presbycusis, hearing loss occurs gradually
over a period of several years. Sensorineural hearing loss can also
be hereditary. A hereditary predisposition may determine the age
at which an individual may begin to experience hearing loss, although
much research has indicated that hearing begins to deteriorate around
the age of 40.
Most sensorineural hearing loss can not be corrected medically or
surgically. In some instances, medications can treat sensorineural
hearing loss, usually cochlear. Treating viral or bacterial infections
via medication such as steroids can improve hearing. Sometimes improving
blood circulation through improved diet and medication can improve
hearing if the loss is caused by circulatory deficiencies. Also,
surgery to remove an acoustic neuroma can restore hearing to that
affected ear. However, most sensorineural hearing loss is permanent.
In most cases of permanent sensorineural hearing loss, hearing
aids can be used to overcome hearing loss and improve communication
and safety. More information about hearing aids and how they can
improve your communication can be found in the “Hearing Aids” section.

This is simply a combination of conductive
and sensorineural hearing loss. For example, a person may have
fluid within the middle ear AND damage to the sensory nerve endings
of the cochlea. A combination of middle ear surgery and use of
hearing aid would be the treatment for this example of mixed hearing
loss.
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Recurrent
ear infections may produce fluid in the middle ear with
subsequent hearing loss. This can cause speech delays and
learning deficits that can be devastating to children. |
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If these infections are not responsive
to antibiotics then a tympanostomy (tube insertion) may
be recommended which removes fluid behind the ear drum
and reverses hearing loss. |
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